The high concentration of iron in the body increases the risk of developing bowel cancer. And one of the main sources of iron is red meat. As it turned out, the iron negatively affects the gene that normally protects against cancer.
Studies in mice have shown that the likelihood of cancer was highly dependent on iron levels and the work of the APC gene. When this gene was performed incorrectly, mice with high levels of consumption of iron in 2-3 times more often ended up living with the disease. But if the mice consumed little iron, even if the defective gene they did not develop cancer.
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As noted by Professor Owen to Sansum, a defective APC gene is 8 cases of cancer out of every 10. Meat also contains a compound called heme (gives meat red color). It damages the lining of the intestines. And in the process of cooking meat are allocated carcinogenic compounds.
Over time, increases the likelihood that cells in the intestine start to produce a defective gene and to respond to iron in the diet. If the gene does not work, iron accumulates in the pavement. This process involves the wnt signaling pathway, leading to uncontrolled cell division.
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Iron also promotes the growth of cells with defective APC. It was proved that mice without the gene problem, or with a normal working gene did not suffer from cancer, despite the high content of iron in the diet. These animals wnt signaling pathway was inactive.
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